proteus-biosciences代理
简要描述:Proteus BioSciences 代理,Proteus BioSciences 中国,Proteus BioSciences 代理,Proteus BioSciences 公司,Proteus BioSciences 专业代理-上海金畔生物科技有限公司,具体产品信息欢迎电询:021-50837765
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世界*实验材料供应商Proteus BioSciences 上海金畔生物为其中国代理,Proteus BioSciences 在一直是行业的*,一直为广大科研客户提供zui为优质的产品和服务,上海金畔生物一直秉承为中国科研客户带来的产品,的服务, Proteus BioSciences 就是为了给广大科研客户带来更加完善的产品和服务,您的满意将是我们zui大的收获
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加拿大Proteus BioSciences www.proteus-biosciences.com
Proteus BioSciences was founded in 2002 to develop innovative products to advance proteomic research. We specialize in discovering peptides and proteins with novel biological properties, designing and developing enzyme activity assays for diverse screening applications, and producing innovative protein analysis tools for biomarker identification and characterization.
With extensive research and product development expertise, academic and industrial biotechnology experience, and a solid business development and technology licensing track record, our efforts have contributed to the the publication of hundreds of peer-reviewed scientific articles and the development of thousands of life science research products. These products support studies in numerous fields including biochemistry, cell and molecular biology, genetics, immunology, neuroscience, and virology. We established and continue to collaborate with a network of over 500 scientific investigators worldwide and we intend to continue encouraging these relationships in the future to improve life science, biomedical and health care research.
Proteus BioSciences was founded in 2002 to discover peptides and proteins with novel biological properties, design and develop enzyme activity assays for diverse screening applications, and produce innovative protein analysis tools. Proteus BioSciences’ products support research in such diverse fields as biochemistry, cell & molecular biology, genetics, immunology, neuroscience and virology.
The Proteus BioSciences product range includes:
Antibodies
Enzyme activity assays
Proteins
Protein analysis
Services offered include production of polyclonal antisera, immunochemistry and purification of fusion-tagged protein.
Myosin-VIIa
Rabbit Polyclonal Antibody
PRODUCT #: 25-6790
DESCRIPTION: Myosin-VIIa is one of the unconventional members of the myosin molecular motor superfamily that move along filamentous actin. The protein is expressed primarily in the inner ear, retina and testis. Defects in the myosin-VIIa gene (MYO7A) are responsible for Usher syndrome, characterized by hearing impairment, lack of vestibular function and progressive retinal degeneration.MYO7A mutations are are responsible for deafness in shaker-1 mice and in other model systems. In the inner ear, myosin-VIIa is required for assembly of stereocilia, the sensory structures necessary for detecting sound signals.
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SPECIES CROSS-REACTIVITY:
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Human, mouse, rat, pig, avian, amphibian
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APPLICATIONS/DILUTIONS: |
IF (5-10 ug/ml) WB (0.5-1.0 ug/ml)
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SOURCE: |
Rabbits were immunized with amino acids 880-1077 from the tail region of human myosin-VIIa.
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FORM/STORAGE: |
50 ug (0.5 mg/ml) of affinity purified IgG with 50% glycerol, 0.01% sodium azide and 1.0 mg/ml BSA. Store at -20o C. Avoid multiple freeze/thaw cycles.
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References: Hasson, T. et al. (1995), Proc. Natl. Acad. Sci. 92:9815-9819. Hasson, T. et al. (1997), Journal of Cell Biology 137:1287-1307. Hasson, T. et al. (1997), Cell Motil. Cytoskel. 37:127-138. Hasson, T. et al. (2005), Cell Motil. Cytoskel. 62:13-26.
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DESCRIPTION: Myosin-VIIa is one of the unconventional members of the myosin molecular motor superfamily that move along filamentous actin. The protein is expressed primarily in the inner ear, retina and testis. Defects in the myosin-VIIa gene (MYO7A) are responsible for Usher syndrome, characterized by hearing impairment, lack of vestibular function and progressive retinal degeneration.MYO7A mutations are are responsible for deafness in shaker-1 mice and in other model systems. In the inner ear, myosin-VIIa is required for assembly of stereocilia, the sensory structures necessary for detecting sound signals.
|
SPECIES CROSS-REACTIVITY:
|
Human, mouse, rat, pig, avian, amphibian
|
APPLICATIONS/DILUTIONS:
|
IF (5-10 ug/ml) WB (0.5-1.0 ug/ml)
|
SOURCE:
|
Rabbits were immunized with amino acids 880-1077 from the tail region of human myosin-VIIa.
|
FORM/STORAGE:
|
50 ug (0.5 mg/ml) of affinity purified IgG with 50% glycerol, 0.01% sodium azide and 1.0 mg/ml BSA. Store at -20o C. Avoid multiple freeze/thaw cycles.
|
References: Hasson, T. et al. (1995), Proc. Natl. Acad. Sci. 92:9815-9819. Hasson, T. et al. (1997), Journal of Cell Biology 137:1287-1307. Hasson, T. et al. (1997), Cell Motil. Cytoskel. 37:127-138. Hasson, T. et al. (2005), Cell Motil. Cytoskel. 62:13-26.
|
DESCRIPTION: Myosin-VIIa is one of the unconventional members of the myosin molecular motor superfamily that move along filamentous actin. The protein is expressed primarily in the inner ear, retina and testis. Defects in the myosin-VIIa gene (MYO7A) are responsible for Usher syndrome, characterized by hearing impairment, lack of vestibular function and progressive retinal degeneration.MYO7A mutations are are responsible for deafness in shaker-1 mice and in other model systems. In the inner ear, myosin-VIIa is required for assembly of stereocilia, the sensory structures necessary for detecting sound signals.
|
SPECIES CROSS-REACTIVITY:
|
Human, mouse, rat, pig, avian, amphibian
|
APPLICATIONS/DILUTIONS: |
IF (5-10 ug/ml) WB (0.5-1.0 ug/ml)
|
SOURCE: |
Rabbits were immunized with amino acids 880-1077 from the tail region of human myosin-VIIa.
|
FORM/STORAGE: |
50 ug (0.5 mg/ml) of affinity purified IgG with 50% glycerol, 0.01% sodium azide and 1.0 mg/ml BSA. Store at -20o C. Avoid multiple freeze/thaw cycles.
|
References: Hasson, T. et al. (1995), Proc. Natl. Acad. Sci. 92:9815-9819. Hasson, T. et al. (1997), Journal of Cell Biology 137:1287-1307. Hasson, T. et al. (1997), Cell Motil. Cytoskel. 37:127-138. Hasson, T. et al. (2005), Cell Motil. Cytoskel. 62:13-26.
|
DESCRIPTION: Myosin-VIIa is one of the unconventional members of the myosin molecular motor superfamily that move along filamentous actin. The protein is expressed primarily in the inner ear, retina and testis. Defects in the myosin-VIIa gene (MYO7A) are responsible for Usher syndrome, characterized by hearing impairment, lack of vestibular function and progressive retinal degeneration.MYO7A mutations are are responsible for deafness in shaker-1 mice and in other model systems. In the inner ear, myosin-VIIa is required for assembly of stereocilia, the sensory structures necessary for detecting sound signals.
|
SPECIES CROSS-REACTIVITY:
|
Human, mouse, rat, pig, avian, amphibian
|
APPLICATIONS/DILUTIONS: |
IF (5-10 ug/ml) WB (0.5-1.0 ug/ml)
|
SOURCE: |
Rabbits were immunized with amino acids 880-1077 from the tail region of human myosin-VIIa.
|
FORM/STORAGE: |
50 ug (0.5 mg/ml) of affinity purified IgG with 50% glycerol, 0.01% sodium azide and 1.0 mg/ml BSA. Store at -20o C. Avoid multiple freeze/thaw cycles.
|
References: Hasson, T. et al. (1995), Proc. Natl. Acad. Sci. 92:9815-9819. Hasson, T. et al. (1997), Journal of Cell Biology 137:1287-1307. Hasson, T. et al. (1997), Cell Motil. Cytoskel. 37:127-138. Hasson, T. et al. (2005), Cell Motil. Cytoskel. 62:13-26.
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上海金畔生物科技有限公司